摘要
记忆是生活的一个相关部分，没有它，人类的生活就会变得悲惨。随着全球老年人口的大量增加，时间已经集中在尾端生命阶段疾病上。阿尔茨海默病（AD）是这类疾病之一，其起源是神秘的，由于认知的不可挽回的损害而严重影响生命的后期，其特征在于存在神经毒性淀粉样蛋白-β（Aβ）斑块和过度磷酸化的Tau蛋白。纤维缠结。现有的治疗方案主要集中在通过靶向继发于AD病理学的神经递质来缓解症状。植物来源的许可药物加兰他敏和石杉碱甲由于其对轻度至中度AD的AChE抑制作用而被广泛研究。尽管许多研究已证明AChEIs作为优选的症状缓解剂的功效，但它们不能提供长期保护。预计AD的下一代药物将改变作为疾病病程基础的各种因素，并具有症状性益处。由于AD涉及复杂的病理学，除了导致产生毒性斑块和神经原纤维缠结的事件之外，必须考虑几种分子差异因素。虽然几种草药已经显示出神经保护作用，但我们已经提到了已经通过实验测试针对不同阿尔茨海默氏病病理模型的植物成分。研究人员需要考虑这些植物成分的进一步药物开发过程，使其在临床上可行，这是目前的一个空白。
关键词: 阿尔茨海默病，β淀粉样蛋白，植物成分，传统医学和神经发生，AChEIs，加兰他敏和石杉碱甲。
图形摘要
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