Abstract
As part of a host-pathogen coevolution, plants have developed very specific resistance proteins (R proteins), which directly or indirectly recognize Avr proteins and thus activate host resistance, or effector-triggered immunity (ETI). ETI can be thought of as an enhanced version of PTI and is often described as leading to localized programmed cell death in infected tissue: the hypersensitive response (HR). This chapter aims to clarify what we know and to identify areas that require further investigation.
Keywords: Effector-triggered Immunity (ETI), Effector recognition, Gene-for Gene resistance, Host cultivar, Hypersensible reaction (HR), Nucleotide Binding Site domain (NBS), Pathogen race, Pathogen effector.
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