DNA Damage Response as a Pharmacological Target for Cancer and Infectious Diseases

Tomasz       Poplawski

doi:10.2174/092986732608190516092613

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[1] 
Rozpędek, W.; Pytel, D.; Nowak-Zduńczyk, A.; Lewko, D.; Wojtczak, R.; Diehl, J.A.; Majsterek, I. Breaking the DNA damage response via serine/threonine kinase inhibitors to improve cancer treatment. Curr. Med. Chem.,  2019, 26(8), 1425-1445.
[2] 
Toma, M.; Skorski, T.; Sliwinski, T. DNA double strand break repair - related synthetic lethality. Curr. Med. Chem.,  2019, 26(8), 1446-1482.
[3] 
Kopa, P.; Macieja, A.; Galita, G.; Witczak, Z.J.; Poplawski, T. DNA Double Strand Breaks Repair Inhibitors: Relevance as Potential New Anticancer Therapeutics. Curr. Med. Chem.,  2019, 26(8), 1483-1493.
[4] 
Minias, A.; Brzostek, A.; Dziadek, J. Targeting DNA repair systems in antitubercular drug development. Curr. Med. Chem.,  2019, 26(8), 1494-1505.

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DOI https://dx.doi.org/10.2174/092986732608190516092613	Print ISSN 0929-8673
Publisher Name Bentham Science Publisher	Online ISSN 1875-533X

Current Medicinal Chemistry

DNA Damage Response as a Pharmacological Target for Cancer and Infectious Diseases

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