Generic placeholder image

Current Alzheimer Research

Editor-in-Chief

ISSN (Print): 1567-2050
ISSN (Online): 1875-5828

Editorial

Inhibition of Store-Operated Calcium Entry in Alzheimer’s Disease and the Associated Mechanisms

Author(s): Shengzhou Wu*

Volume 17, Issue 12, 2020

Page: [1055 - 1056] Pages: 2

DOI: 10.2174/156720501712210129111258

[1]
Sun S, Zhang H, Liu J, et al. Reduced synaptic STIM2 expression and impaired store-operated calcium entry cause destabilization of mature spines in mutant presenilin mice. Neuron 2014; 82(1): 79-93.
[http://dx.doi.org/10.1016/j.neuron.2014.02.019] [PMID: 24698269]
[2]
Yoo AS, Cheng I, Chung S, et al. Presenilin-mediated modulation of capacitative calcium entry. Neuron 2000; 27(3): 561-72.
[http://dx.doi.org/10.1016/S0896-6273(00)00066-0] [PMID: 11055438]
[3]
Leissring MA, Akbari Y, Fanger CM, et al. Capacitative calcium entry deficits and elevated luminal calcium content in mutant presenilin-1 knockin mice. J Cell Biol 2000; 149(4): 793-8.
[http://dx.doi.org/10.1083/jcb.149.4.793] [PMID: 10811821]
[4]
Bojarski L, Pomorski P, Szybinska A, et al. Presenilin-dependent expression of STIM proteins and dysregulation of capacitative Ca2+ entry in familial Alzheimer’s disease. Biochim Biophys Acta 2009; 1793(6): 1050-7.
[http://dx.doi.org/10.1016/j.bbamcr.2008.11.008] [PMID: 19111578]
[5]
Tu H, Nelson O, Bezprozvanny A, et al. Presenilins form ER Ca2+ leak channels, a function disrupted by familial Alzheimer’s disease-linked mutations. Cell 2006; 126(5): 981-93.
[http://dx.doi.org/10.1016/j.cell.2006.06.059] [PMID: 16959576]
[6]
Tong BC, Lee CS, Cheng WH, Lai KO, Foskett JK, Cheung KH. Familial Alzheimer’s disease-associated presenilin 1 mutants promote γ-secretase cleavage of STIM1 to impair store-operated Ca2+ entry. Sci Signal 2016; 9(444): ra89.
[http://dx.doi.org/10.1126/scisignal.aaf1371] [PMID: 27601731]
[7]
Kuang XL, Liu Y, Chang Y, et al. Inhibition of store-operated calcium entry by sub-lethal levels of proteasome inhibition is associated with STIM1/STIM2 degradation. Cell Calcium 2016; 59(4): 172-80.
[http://dx.doi.org/10.1016/j.ceca.2016.01.007] [PMID: 26960935]
[8]
Zhou J, Song J, Wu S. Autophagic degradation of stromal interaction molecule 2 mediates disruption of neuronal dendrites by endoplasmic reticulum stress. J Neurochem 2019; 151(3): 351-69.
[http://dx.doi.org/10.1111/jnc.14712] [PMID: 31038732]
[9]
Zhu ZD, Yu T, Liu HJ, Jin J, He J. SOCE induced calcium overload regulates autophagy in acute pancreatitis via calcineurin activation. Cell Death Dis 2018; 9(2): 50.
[http://dx.doi.org/10.1038/s41419-017-0073-9] [PMID: 29352220]
[10]
McLarnon JG. Microglial store-operated calcium signaling in health and in Alzheimer’s disease. Curr Alzheimer Res 2020; 17(12): 1055-63.
[PMID: 33463465]
[11]
Fine tuning of intracellular Ca2+ content by pharmacological agents - a strategy to prevent synapse loss in Alzheimer disease hippocampal neurons. Curr Alzheimer Res 2020; 17(12): 1064-70.
[12]
Huang AS, Tong BC, Wu AJ, et al. Rectifying attenuated store-operated calcium entry as a therapeutic approach for Alzheimer’s disease. Curr Alzheimer Res 2020; 17(12): 1071-87.
[PMID: 33463469]
[13]
Zhou J, Wu S. Impairment of store-operated calcium entry: implications in Alzheimer’s neurodegeneration. Curr Alzheimer Res 2020; 17(12): 1088-94.
[PMID: 33463466]

© 2024 Bentham Science Publishers | Privacy Policy