Abstract
Due to an oversight of the corresponding author, the list of authors was originally published incorrectly in the manuscript entitled “Therapeutic Approaches to Modulating Glutathione Levels as a Pharmacological Strategy in Alzheimer’s Disease”, published in Current Alzheimer Research, volume 12, no. 4, 2015, pp. 298-313.
Due to this oversight, the correct first author was omitted from the original byline. The correct list of authors, with the true first author, is provided below:
Authors: Peter Cao, Nady Braidy, Martin Zarka, Jeffrey Welch and Wallace Bridge.
Affiliation: School of Biotechnology and Biomolecular Sciences, Faculty of Science, University of New South Wales, Syd-ney, Australia, NSW 2052.
Current Alzheimer Research
Title:Erratum: Therapeutic Approaches to Modulating Glutathione Levels as a Pharmacological Strategy in Alzheimer’s Disease
Volume: 13 Issue: 10
Author(s):
Affiliation:
Abstract: Due to an oversight of the corresponding author, the list of authors was originally published incorrectly in the manuscript entitled “Therapeutic Approaches to Modulating Glutathione Levels as a Pharmacological Strategy in Alzheimer’s Disease”, published in Current Alzheimer Research, volume 12, no. 4, 2015, pp. 298-313.
Due to this oversight, the correct first author was omitted from the original byline. The correct list of authors, with the true first author, is provided below:
Authors: Peter Cao, Nady Braidy, Martin Zarka, Jeffrey Welch and Wallace Bridge.
Affiliation: School of Biotechnology and Biomolecular Sciences, Faculty of Science, University of New South Wales, Syd-ney, Australia, NSW 2052.
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Cite this article as:
, Erratum: Therapeutic Approaches to Modulating Glutathione Levels as a Pharmacological Strategy in Alzheimer’s Disease, Current Alzheimer Research 2016; 13 (10) . https://dx.doi.org/10.2174/156720501310160825221218
DOI https://dx.doi.org/10.2174/156720501310160825221218 |
Print ISSN 1567-2050 |
Publisher Name Bentham Science Publisher |
Online ISSN 1875-5828 |
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Neuroinflammation is an invariable hallmark of chronic and acute neurodegenerative disorders and has long been considered a potential drug target for Alzheimer?s disease (AD) and dementia. Significant evidence of inflammatory processes as a feature of AD is provided by the presence of inflammatory markers in plasma, CSF and postmortem brain ...read more
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