[1]
Deryabina EG, Yakornova GV, Pestryaeva LA, Sandyreva ND. Perinatal outcome in pregnancies complicated with gestational diabetes mellitus and very preterm birth: case-control study. Gynecol Endocrinol 2016; 32: 52-5.
[2]
Guzmán-Gutiérrez E, Arroyo P, Salsoso R, et al. Role of insulin and adenosine in the human placenta microvascular and macrovascular endothelial cell dysfunction in gestational diabetes mellitus. Microcirculation 2014; 21: 26-37.
[3]
Huynh J, Yamada J, Beauharnais C, et al. Type 1, type 2 and gestational diabetes mellitus differentially impact placental pathologic characteristics of uteroplacental malperfusion. Placenta 2015; 36: 1161-6.
[4]
Jarmuzek P, Wielgos M, Bomba-Opon D. Placental pathologic changes in gestational diabetes mellitus. Neuroendocrinol Lett 2015; 36: 101-5.
[5]
Zong S, Li C, Luo C, et al. Dysregulated expression of IDO may cause unexplained recurrent spontaneous abortion through suppression of trophoblast cell proliferation and migration. Sci Rep 2016; 27: 19916.
[6]
Tian FJ, Qin CM, Li XC, et al. Decreased stathmin-1 expression inhibits trophoblast proliferation and invasion and is associated with recurrent miscarriage. Am J Pathol 2015; 185: 2709-21.
[7]
Cao YL, Jia YJ, Xing BH, et al. Plasma microRNA-16-5p, -17-5p and -20a-5p: Novel diagnostic biomarkers for gestational diabetes mellitus. J Obstet Gynaecol Res 2017; 43: 974-81.
[8]
Shi Z, Zhao C, Guo X, et al. Differential expression of microRNAs in omental adipose tissue from gestational diabetes mellitus subjects reveals miR-222 as a regulator of ERα expression in estrogen-induced insulin resistance. Endocrinology 2014; 155: 1982-90.
[9]
Muralimanoharan S, Maloyan A, Myatt L. Mitochondrial function and glucose metabolism in the placenta with gestational diabetes mellitus: role of miR-143. Clin Sci (Lond) 2016; 130: 931-41.
[10]
Kotlabova K, Doucha J, Hromadnikova I. Placental-specific microRNA in maternal circulation--identification of appropriate pregnancy-associated microRNAs with diagnostic potential. J Reprod Immunol 2011; 89: 185-91.
[11]
Lamadrid-Romero M, Solís KH, Cruz-Reséndiz MS, et al. Central nervous system development-related microRNAs levels increase in the serum of gestational diabetic women during the first trimester of pregnancy Neurosci Res 2017. 10:
pii: S0168-0102(17)30246-8
[12]
Li J, Song L, Zhou L, et al. A MicroRNA Signature in Gestational Diabetes Mellitus Associated with Risk of Macrosomia. Cell Physiol Biochem 2015; 37: 243-52.
[13]
Lu Y, Heng X, Yu J, et al. miR-137 regulates the migration of human umbilical vein endothelial cells by targeting ephrin-type A receptor 7. Mol Med Rep 2014; 10: 1475-80.
[14]
Li J, Li J, Wei T, et al. Down-Regulation of MicroRNA-137 Improves High Glucose-Induced Oxidative Stress Injury in Human Umbilical Vein Endothelial Cells by Up-Regulation of AMPKα1. Cell Physiol Biochem 2016; 39: 847-59.
[15]
Peng HY, Li MQ, Li HP. High glucose suppresses the viability and proliferation of HTR-8/SVneo cells through regulation of the miR-137/PRKAA1/IL-6 axis. Int J Mol Med 2018; 42: 799-810.
[16]
Lu TM, Lu W, Zhao LJ. MicroRNA-137 Affects Proliferation and Migration of Placenta Trophoblast Cells in Preeclampsia by Targeting ERRα. Reprod Sci 2016; 6: 1933719116650754.
[17]
Zhao L, Li J, Li ZL, et al. Circulating irisin is lower in gestational diabetes mellitus. Endocr J 2015; 62: 921-6.
[18]
Erol O, Erkal N, Ellidağ HY, et al. Irisin as an early marker for predicting gestational diabetes mellitus: a prospective study. J Matern Fetal Neonatal Med 2016; 29: 3590-5.
[19]
Staiger H, Böhm A, Scheler M, et al. Common genetic variation in the human FNDC5 locus, encoding the novel muscle-derived ‘browning’ factor irisin, determines insulin sensitivity. PLoS One 2013; 8: e61903.
[20]
Shi X, Lin M, Liu C, et al. Elevated circulating irisin is associated with lower risk of insulin resistance: association and path analyses of obese Chinese adults. BMC Endocr Disord 2016; 16: 44.
[21]
Han CS, Herrin MA, Pitruzzello MC, et al. Glucose and metformin modulate human first trimester trophoblast function: a model and potential therapy for diabetes-associated uteroplacental insufficiency. Am J Reprod Immunol 2015; 73: 362-71.
[22]
Tanisawa K, Taniguchi H, Sun X, et al. Common single nucleotide polymorphisms in the FNDC5 gene are associated with glucose metabolism but do not affect serum irisin levels in Japanese men with low fitness levels. Metabolism 2014; 63: 574-83.
[23]
Floris I, Descamps B, Vardeu A, et al. Gestational diabetes mellitus impairs fetal endothelial cell functions through a mechanism involving microRNA-101 and histone methyltransferase enhancer of zester homolog-2. Arterioscler Thromb Vasc Biol 2015; 5: 664-74.
[24]
Loginov VI, Rykov SV, Fridman MV, Braga EA, et al. Methylation of miRNA genes and oncogenesis. Biochemistry (Mosc) 2015; 80: 145-62.
[25]
Coustan DR, Lowe LP, Metzger BE. The hyperglycemia and adverse pregnancy outcome (HAPO) study: can we use the results as a basis for change? J Matern Fetal Neonatal Med 2010; 23: 204-9.
[26]
Bonomo JA, Guan M, Ng MC, et al. The ras responsive transcription factor RREB1 is a novel candidate gene for type 2 diabetes associated end-stage kidney disease. Hum Mol Genet 2014; 23: 6441-7.
[27]
Franklin RB, Zou J, Costello LC. The cytotoxic role of RREB1, ZIP3 zinc transporter, and zinc in human pancreatic adenocarcinoma. Cancer Biol Ther 2014; 15: 1431-7.
[28]
Barja-Fernández S, Folgueira C, Castelao C, et al. FNDC5 is produced in the stomach and associated to body composition. Sci Rep 2016; 6: 23067.
[29]
Pukajło K, Kolackov K, Łaczmański Ł, et al. Irisin--a new mediator of energy homeostasis. Postepy Hig Med Dosw(Online) 2015; 69: 233-42.
[30]
Srinivasa S, Suresh C, Mottla J, et al. FNDC5 relates to skeletal muscle IGF-I and mitochondrial function and gene expression in obese men with reduced growth hormone. Growth Horm IGF Res 2016; 26: 36-41.
[31]
Liu S, Du F, Li X, et al. Effects and underlying mechanisms of irisin on the proliferation and apoptosis of pancreatic β cells. PLoS One 2017; 12: e0175498.
[32]
Zhu G, Wang J, Song M, et al. Irisin increased the number and improved the function of endothelial progenitor cells in diabetes mellitus mice. J Cardiovasc Pharmacol 2016; 68: 67-73.
[33]
Xie C, Zhang Y, Tran TD, et al. Irisin controls growth, intracellular Ca2+ signals, and mitochondrial thermogenesis in cardiomyoblasts. PLoS One 2015; 10: e0136816.
[34]
Zhang Y, Mu Q, Zhou Z, et al. Protective effect of irisin on atherosclerosis via suppressing oxidized low density lipoprotein induced vascular inflammation and endothelial dysfunction. PLoS One 2016; 11: e0158038.
[35]
Song H, Wu F, Zhang Y, et al. Irisin promotes human umbilical vein endothelial cell proliferation through the ERK signaling pathway and partly suppresses high glucose-induced apoptosis. PLoS One 2014; 9: e110273.
[36]
Han F, Zhang S, Hou N, et al. Irisin improves endothelial function in obese mice through the AMPK-eNOS pathway. Am J Physiol Heart Circ Physiol 2015; 309: H1501-8.